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These results mean that the inhibition of CysLT1 and LTB4 receptors would be a potential and promising new therapeutic method to prevent inflammation and disease progression in RA patients ( Figure 3 )
To further confirmed that TXNDC12 inhibits PAAD cell ferroptosis via GSH metabolism, we used the GSH synthesis inhibitor BSO (PANC-1 1.53 mM, MIA PaCa-2 3.506 mM) and exogenous GSH 500 M to incubate the cells and detected changes in the content of GSH and GSSG while knocking down TXNDC12
Results, logged via weekly weigh-ins and monthly DEXA scans, showed: Body weight: mean change 0.2 kg (95% CI 0.8 to +0.4), not statistically significant
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