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Therefore, a comprehensive understanding of these compensatory mechanisms is imperative for the development of more effective therapeutic strategies that target cancer cells [212]
Given that Bacteroides abundance is typically reduced in patients with COPD, particularly during acute exacerbations (Bowerman et al., 2020), these findings imply that restoration or targeted modulation of this genus could constitute a plausible mechanism by which microbiota-based interventions augment Trp metabolic capacity and regulate disease progression
This framework supports reliable physicochemical performance, including solubility behavior and stability in experimental conditions, enabling consistent results in signaling, metabolic, and biochemical assays
Ann N Y Acad Sci 1446(1):4465
Hence, further strengthening the understanding of the roles of various classes of ncRNAs involved in regulating the common processes in cancer development and fungal pathogenesis could help to design treatment strategies such as ncRNA mimics or antagonist [101]
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