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Calorie restriction in humans inhibits the PI3K/AKT pathway and induces a younger transcription profile

While therapeutic strategies targeting oxidative stress are crucial, their clinical translation has largely represented an unmet promise due to the complexity of redox biology, delivery challenges, and patient heterogeneity

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By simultaneously pushing the accelerator via GHRH receptors and removing the brakes via somatostatin inhibition, the resulting pulse is significantly more potent than either peptide could produce in isolation