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M.ChaudharyN.ChabenneJ
GLP-1, acting on its receptor, has physiological actions on multiple target organs: it increases glucose-stimulated insulin secretion in pancreatic beta cells, reduces glucagon secretion by cells, improves insulin sensitivity and reduces food intake.26 In cellular and murine models GLP-1 promotes cell neogenesis and inhibits their apoptosis.27 Insulin secretion induced by GLP-1 is dependent of the glycemia and therefore does not induce hypoglycemia (Fig
This not only accelerates the healing process but also improves the quality of the healed tissue, reducing the risk of complications such as infections or delayed healing
A similar upregulation in striatal levels of GSTO2 was observed during the presymptomatic phase, concomitant with BDNF downregulation before the increases in dopamine
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