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Unlike GLP-1 receptor agonists, Cagrilintide works through amylin and calcitonin receptors in the brain, offering a separate but complementary pathway for controlling hunger and reducing caloric intake

doi: 10.1002/kjm2.12226 181 ZhangMXueSJYangFXiaoMTangYBWuY

Proposed Mechanism BPC-157 appears to work through multiple pathways: Maintaining vesicular integrity (affecting VMATs and dopamine transporters) Possibly occupying its own vesicular storage sites Competing for dopamine receptor binding Functionally substituting when dopaminergic neurons are lost This last property, essentially filling in for missing dopamine function, would explain why the peptide counteracts both receptor agonists (apomorphine) and antagonists (haloperidol, fluphenazine, clozapine, sulpiride) with equal effectiveness