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This may involve targeting the CBX3/NRF2/GPX2 axis with CBX3 or NRF2 inhibitors or impairing antioxidant buffering systems such as glutathione synthesis (e.g., via GCLC suppression)

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Myocardial scarring is thought to either induce or maintain ventricular arrhythmias in several ways: 1) scarring creates insulating barriers that form alterations in conduction in the fibrotic tissue, resulting in re-entrant wave fronts of excitation

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