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[ref] The AGXT gene encodes the enzyme responsible for breaking down glyoxylate into glycine so that it doesnt get converted into oxalates

Treatment with non-metabolizable glucose analogs and the glycolysis inhibitor 2-deoxyglucose or knockdown of cFLIP abolished the above effects, suggesting that GLUT1 exerts its anti-apoptotic effects through glycolysis [58]

Forward flow depends on adequate removal of homocysteine and especially adenosine

This is completely normal

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