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In pancreatic cancer, the Kirsten rat sarcoma viral oncogene homolog (KRAS) gene reprograms glutamine metabolism by upregulating aspartate transaminase (GOT1) and repressing GLUD1 to increase the NADPH/NADP(+) ratio for stable redox capacity (Son et al., 2013)
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Patients carrying pathogenic mtDNA mutations or rare FAO enzyme deficiencies also represent a niche population for gene-based therapies
Loss of the mitochondrial membrane potential was detected using the lipophilic cationic dye JC-1 that displays potential-dependent accumulation in the mitochondria, thus analysis of JC-1-derived fluorescence signal allows for distinguishing between healthy and apoptotic cells
