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Ultimately, this FOXO4-DRI-mediated mechanism selectively eliminates senescent endothelial cells, which in turn attenuates vascular inflammation and improves endothelial-dependent vasodilation, conferring a significant protective effect on vascular function
L.MenichettiG
Protocols that incorporate inflammation as a primary variable alongside regenerative mechanisms (chronic inflammatory models, oxidative stress research, or age-associated tissue decline) represent the expanded scope of this formulation relative to the Glow base
doi:10.1073/pnas.89.18.8641
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