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This paper systematically reviews the regulation of GA metabolism and immune responses in the hyperuricemia occurring due to FA metabolic disorders and identifies targets for personalized therapy based on the underlying mechanisms ( Disordered FA metabolism further promotes inflammation, and elevation of the uric acid levels, primarily through high levels of FAs, activates the initiation of inflammation

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2b) in male db / db mice following a single intraperitoneal (i.p.) injection