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Collectively, these findings indicate that reduced adipocyte mitochondrial CI activity during obesity is a key driver of visceral WAT metabolic dysfunction and subsequent systemic impairment in metabolic health

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Through promoting collagen synthesis, angiogenesis, and cell migration, BPC-157 supports structural healing in tissues damaged by Lyme-related inflammation

Similarly, 4-aminobiphenyl, metabolized by hepatic CYP1A2, produces reactive intermediates that form DNA adducts in hepatocytes, increasing the mutational burden [27]