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NRF2 is considered a central regulator of the cellular responses to changes in redox status but is also an important regulator of an array of processes that includes mitochondrial function, xenobiotic metabolism, carbohydrate, lipid, and iron metabolism, and DNA repair

We also observed the ability of GLP-1(2836) to increase cAMP production through bicarbonate-responsive (i.e., probenicid-inhibited) sAC in human caECs

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Mitochondrial DNA deletions and neurodegeneration in multiple sclerosis

When experiments include receptor-selective controls, they can disentangle which pathway drives each measured effect, improving interpretability