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Additionally, reduced expression of one 25-hydroxylase protein (CYP27A1
Riboflavin is required for the production of FAD and FMN
Furin cleavage exposes the S2 subunit for further processing by the host serine proteases for subsequent viral entry 72,73
Furthermore, excessive ROS generation can disrupt nitric oxide (NO) homeostasis by reducing endothelial nitric oxide synthase (eNOS) activity, while upregulating inducible nitric oxide synthase (iNOS), leading to impaired vasodilation and exacerbated venous stasis [11]
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