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doi: 10.1097/BRS.0b013e3181ccb1b3 28 BratDJGianniniCScheithauerBWBurgerPC

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Artificial overexpression of GPx1 showed an increased capacity to rescue breast cancer cells from the cell cycle arrest caused by hyperoxic stress, indicating the involvement of both peroxide-derived free radicals and nonperoxide-derived species during the deleterious process [84]