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In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis

B-vitamins act as coenzymes in numerous metabolic pathways, including carbohydrate and fat metabolism, neurotransmitter production, and DNA synthesis

All prescriptions are written at the sole discretion of the licensed provider

NAD+ Injections NAD+ Injections NAD is essential to cellular health and function, but levels naturally decline as we age

Adequate B12 allows normals RBC formation (preventing anemia-related fatigue) and keeps nerves myelinated