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GHRH activates its own receptor (GHRH-R) on the same somatotroph cells, primarily through cAMP-protein kinase A signaling
Together, our work establishes SLC33A1-mediated GSSG export as a key mechanism for ER redox homeostasis and protein maturation
Intriguingly, these MTs often act as tumor suppressor genes in CRC
Some patients experience a temporary worsening of symptoms during the early phase of treatment as mycotoxins are mobilized from tissue storage
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