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The effect of lowering fasting glucose level and glycated hemoglobin is generally greater with long-acting agents, and the effect of lowering postprandial glucose level is more evident with short-acting agents
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Since oxidative stress is the main cause of APAP-induced hepatotoxicity, sulforaphane protects the liver from APAP overload toxicity by activating the Nrf2 pathway and increasing the endogenous antioxidant response (Schmidt, 1984)
Mller J, Jrgensen JO, Mller N, et al
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