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Reactive intermediates resulting from lipid peroxidation (4hydroxynonenal), nucleotide peroxidation (adenine propenal) or catecholamine peroxidation (aminochrome, dopachrome, adrenochrome) are particularly inactivated by GSTs (DagninoSubiabre et al., 2000)

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These findings suggest that semaglutide protects against A-induced neurotoxicity by promoting autophagy and suppressing apoptosis

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