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Ferroptosis activation in TGF--treated HK-2 cells is suppressed by GPX4 overexpression Our in vivo studies revealed the role of GPX4 overexpression in attenuating TEC ferroptosis and fibrotic changes during UUO

Beta-adrenergic agonists can activate sodium and chloride trafficking across the alveolar epithelium by stimulating the -2 receptor to upregulate cAMP, which re-creates the osmotic gradient for water reabsorption from the damaged ARDS alveoli (Perkins et al., 2008)

Gao Y, Wang C, Wang K, He C, Hu K, Liang M

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