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The re-introduction of O 2 in the ischemia or long tissue hypoxia can cause aditional tissue injury (re-oxygenation injury) which is partiatly mediated by oxygen radicals (Halliwell and Gutteridge, 1989)
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In that sense, while still representing a challenging goal to be achieved, the identification of specific prognostic biomarkers and also those for the evaluation of therapeutic efficacy in ALS will have a fundamental role in designing and monitoring new treatment strategies in the complex scenario of the neurodegenerative process
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