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Oncolytic Newcastle disease virus induced degradation of YAP through E3 ubiquitin ligase PRKN to exacerbate ferroptosis in tumor cells
The compounds bind to entirely different receptors (beta-3 adrenergic vs GLP-1/GIP)
GaoZYinJZhangJWardREMartinRJLefevreMet al
The results indicate that Gipr -expressing cells in the AP underlie the ability of GIPR agonists to reduce food intake in lean and DIO mice and, taken together with the previous report that GABAergic Gipr KO mice do not exhibit reduced food intake in response to acyl-GIP, suggest that subpopulations of GABAergic Gipr -expressing neurones located in the AP mediate both the appetite-suppressing and anti-aversive effects of exogenous GIPR agonists 6
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