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Altogether, it seems that overexpressing IGF-1 plays a dual role in prevention of oxidative SIPS in hepatocytes: on the one hand, in cytoplasm, IGF-1 activates the PI3K/AKT1 pathway to inhibit p53 nuclear translocation, mediated by the AKT1p53 interaction

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Wong, H.W.G., Elwell, J.H., Oberley, L.W

Optimizing your GHK-Cu hair protocol Generic protocols provide starting points

Muscle-specific deletion of carnitine acetyltransferase compromises glucose tolerance and metabolic flexibility