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OL loss in progressive MS In contrast to acute lesions, there is consensus of significant oligodendroglial cell loss in the center of chronic active lesions as indicated by neuropathological observations of post-mortem tissue 8 and single nuclei sequencing 47
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[1] [2] It is a dual glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor agonist
Schoenenberger, P., Monnier, M., & Gaillard, R
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