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It is classified as a potent antioxidant with demonstrated effects on lipid metabolism, insulin sensitivity, and inflammation in both animal and human studies

CD4 + , CD8 + , and T cells drive inflammation and activate HSCs, which in turn regulate T cell differentiation and proliferation

Analysis of ferroptosis-related protein expression revealed that ginkgolide B (GB) treatment significantly upregulated NRF2, a key regulator of ferroptosis, in both liver tissue and hepatocyte models

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[39] The second factor is the presence of proteins in the liver and bile that either promote or inhibit cholesterol crystallization into gallstones