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2079-A NASH CIRRHOSIS: DISTINCT PHENOTYPICAL DIFFERENCES, MORE SYSTEMIC INFLAMMATION, AND MORE FRAIL THAN VIRAL-RELATED CIRRHOSIS Yu JUN Wong 1,2 , Heng Yi Tan 3 , Siew Yoon Yap 4 , Geraldine Lim 4 , Seok Hwee Khoo 4 , Jessica Tan 1 , Rahul Kumar 1,2 , Joan Khoo 5 , Rosario Helen Barbara 6 and Paul Edward Hutchinson 3 , (1)Department of Gastroenterology & Hepatology, Changi General Hospital, (2)Duke-Nus Medical School, Singhealth, (3)Flow Cytometry Laboratory, Life Sciences Institute, National University of Singapore, (4)Clinical Trial Research Unit, Changi General Hospital, (5)Department of Endocrinology, Changi General Hospital, (6)Department of Geriatric Medicine, Changi General Hospital Background: NASH is replacing viral hepatitis as the fastest-growing etiology of liver cirrhosis globally with limited therapeutic options

Prior to mixing, allow the vial to reach ambient room temperature to prevent condensation

Ann Rheum Dis (2016) 75(2):4229

Using this phenotypical characterization, most of FoB and MZB are thought to derive from TrB1 that have transitioned to TrB2 61,62

Over-supplementation without need can cause interactions with other nutrients or mild digestive issues