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The gene set enrichment analysis (GSEA) of gene ontology (GO) pathways of RNA sequencing data revealed that T315I-Bcr-Abl cells have an increase in ATF4-mediated integrated stress response (ISR) signaling (Supplementary Fig

[1] [3] The medication works by mimicking the incretin hormone GLP-1, which stimulates insulin secretion in a glucose-dependent manner, suppresses glucagon release, slows gastric emptying, and reduces appetite through central nervous system pathways

Therefore, mortality findings are reported descriptively and should not be interpreted as evidence of treatment effect

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