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On the other hand, treatment of cancer cells with the anti-IGF-IR mAb cixutumumab paradoxically triggers cancer cells to produce IGF2, which in turn recruits macrophages and fibroblasts to promote angiogenesis and metastasis thereby hampering the efficacy of cixutumumab [190]
Every protocol is designed for a specific physiological outcome
Presented for research literature review only Najafi M et al
10.1124/pr.57.4.2 131 KassahunK.PearsonP
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