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Further mechanistic investigations determined that the elevation of intracellular GSSG levels resulted in NF-B inhibition by facilitating glutathionylated IKK- (IKK--SSG) formation, leading to enhanced TNF hepatotoxicity
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2019), or to support regeneration of GSH oxidized by other compounds such as superoxide (Polle 2001), as well as some of the thiol peroxidases discussed further below
Effects of nilvadipine on cerebral blood flow in patients with alzheimer disease
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