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Demonstrating peripheral effects by suppressing T-cell-mediated inflammation via GLP-1 receptor activation in intestinal lymphocytes, and central effects by mediating Toll-like receptor (TLR)-induced anti-inflammatory responses through GLP-1 receptor signaling in the brain [39]

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BBB disruption: Pro-inflammatory cytokines, generated from systemic immune activation, compromise BBB integrity through mechanisms involving tight junction protein degradation and increased transcytosis (Sait and Day, 2024)

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