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BPC-157 and TB-500 work through complementary pathways: BPC-157 through VEGF-driven angiogenesis and growth hormone receptor activation, TB-500 through actin regulation and cellular migration

1 Introduction Ulcerative colitis (UC) is a chronic, relapsing form of inflammatory bowel disease (IBD) characterized by immune system dysfunction and pathological overactivation of mucosal immune responses, leading to persistent inflammation and subsequent colonic mucosal injury (Tsujii et al., 2022)

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