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Collectively, these findings underscore a pathogenic synergy wherein -syn aggregation and lipid peroxidation co-amplify each other, culminating in ferroptotic cell death and neurotoxicity
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In contrast, the total H3 acetylation in the liver was significantly enhanced after NaB intervention in vivo, and the total H3 acetylation was also obviously enhanced in vitro in HepG2 cells after NaB treatment, independent of GPR43 or GPR109a
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