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Although the specific molecular mechanisms through which obesity promotes carcinogenesis remain not fully elucidated, epidemiological studies indicate that approximately 48% of malignant tumors can be attributed to obesity, and obesity serves as an independent risk factor for poor prognosis in cancer patients [54]
have anti-inflammatory, anti-ischemic and anti-fibrotic effects and inhibit myocardiocyte apoptosis.29,32,42 More recently, it has been reported that they also reduce epicardial fat.43 They also inhibit platelet aggregation and thrombosis.44 In the lungs, rGLP-1 activation conferred protection against lipopolysaccharide-induced acute lung injury.45 In adipose tissue they favor adipocyte differentiation, decreases lipid deposition in white fat and facilitate its conversion to brown fat, as well as thermogenesis and energy consumption29,32,4648
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