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In fibrosis-related contexts, it has also been described as a regulator of pathways related to tissue remodeling[2]

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Thus, the observed c-Fos activation pattern elicited by semaglutide in this study is consistent with a brainstem-to-amygdala circuit engaged in appetite-regulating pathways

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Together, these data demonstrate that the abundance of triglycerides in Gclc KO mice is lower, especially in circulating pools