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This endogenous release mechanism positions GHK-Cu as a natural damage signal that initiates repair cascades
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impact on fertility
The latter is referred to as the senesence associated secretory phenotype (SASP) and this is considered to be driving many aspects of functional decline
Animals also presented a reduction of tight junction protein ZO-1, suggesting other effects into BBB, and exhibited augment in lipid peroxidation in the brain [29]
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