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The compound's triple agonist mechanism may be considered sufficiently comprehensive that additional combinations aren't prioritized in the development pipeline
The hypothesis that deficient serotonergic neurotransmission contributes to the pathogenesis of MDD originated from clinical observations that amine-depleting drugs induced depressive states, whereas monoamine oxidase inhibitors, which prevent the breakdown of serotonin (5-HT), alleviated depressive symptoms [4, 5]
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Protection against radiation-induced oxidative stress in cultured human epithelial cells by treatment with antioxidant agents
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