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It manifests as increased tissue turgor and musculoskeletal discomfort resulting in a variety of adverse reactions (e.g

On one hand, the central inflammatory component may be triggered by the immunologic response to infectious agents (Glassford 2017)

Moreover, fibrosis markers (HA, PC III, LN) significantly decreased in the glutathione-treated patients, suggesting improved liver tissue health

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Finally, reduced excitotoxicity and improved overall handling of glutamate could be achieved through improved metabolism (e.g., increased ATP production) in astrocytes, which are responsible for the uptake of glutamate from the synaptic cleft (113)