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In normal physiology, the majority of circulating IGF-1 is bound to IGFBP-3, forming a large ternary complex with the acid-labile subunit that prevents rapid clearance and buffers free IGF-1 within a tightly controlled range
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This coordinated action results in faster healing, reduced scar tissue formation, and improved functional recovery compared to single-peptide approaches
Nat Genet 2010
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