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Central administration into brain ventricles produces more pronounced effects on food intake and body weight than peripheral administration, confirming the importance of hypothalamic MC4R activation
levels can lead to mitochondrial permeability transition pore (mPTP) opening, cytochrome C release, and activation of the caspase cascade (282, 283)
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Beyond their islet effects, incretins exert pleiotropic actions on the brain (appetite and satiety regulation), gastrointestinal tract (delayed gastric emptying), adipose tissue (lipolysis modulation), and liver (inhibition of hepatic glucose output) (13)
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