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While tirzepatide does not shrink the heart, patients should be aware of important cardiovascular considerations when initiating and continuing therapy
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Such molecules can bind to proteins (which contain hundreds or even thousands of amino acids), altering these proteins function, and therefore serving as biochemical messengers
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The underlying molecular mechanism is that tumours driven by BRAF V600E exhibit high extracellular signal-regulated kinase phosphorylation, which activates the mitogen-activated protein kinase (MAPK) signalling pathway, leading to unregulated cell proliferation and a poor prognosis [72]
