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In the case of high autophagic flux, ferritin is degraded, releasing more Fe 2+ , which further promotes the Fenton reaction and lipid peroxidation, thereby exacerbating ferroptosis and causing greater membrane damage
GSH is a critically important molecule in both direct reactions with oxidants as well as several enzymatic detoxification pathways
Loss of SQLE expression did not affect host cell viability (Fig S3C), and Cryptosporidium growth and development was indeed significantly enhanced in SQLE -KO cells compared to WT controls (Fig 3A)
They reduce mitochondrial oxidative damage by restricting mitochondrial ROS generation and increasing mitochondrial antioxidant systems such as mitochondrial SOD
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