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Animal-model evidence consistently shows local effects: angiogenesis at the injury site (VEGF upregulation), growth-factor signaling (EGF, FGF), nitric oxide pathway modulation, and accelerated tendon-to-bone interface healing in rat Achilles transection studies
When it fails, the consequences can be severe
The peptides interaction with molecular pathways, particularly those involving nitric oxide and VEGF, underscores its unique capabilities in promoting tissue repair and healing
They can eliminate it entirely, leaving you injecting an expensive solution that does nothing
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