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Table 2 summarizes some of the variants reported to be associated with neonatal hyperbilirubinemia
10.1080/09513590.2024.2302402 136 ZhuangC.LuoX.WangW.SunR.QiM.YuJ
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Archived from the original on August 4, 2009
Several complex signal transduction pathways and cellular processes have been implicated in APAP hepatotoxicity, including those involving c-Jun N-terminal kinase, Nuclear factor E2-related factor 2 (Nrf2), p53, endoplasmic reticulum stress, autophagy, and sterile inflammation (Chowdhury et al
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