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Altered brain microRNA biogenesis contributes to phenotypic deficits in a 22q11-deletion mouse model
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doi: 10.1097/HJH.0000000000000773 282 ZhangX.ZhivakiD.Lo-ManR
This means that it binds to the same receptors as does endogenous ghrelin (the GHSR-1a receptors) and that it stimulates the cells in the pituitary gland to release more GH without raising ACTH or cortisol levels [3]
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