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S100B and S100A6 differentially modulate cell survival by interacting with distinct RAGE (receptor for advanced glycation end products) immunoglobulin domains
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This risk can be mitigated through Fc engineering, such as introducing point mutations (e.g., L234A/L235A, or N297A) to reduce or ablate FcR binding, thereby minimizing Fc-mediated, antigen-independent uptake [23]
Dodatkowo replikacja gwnego efektu (indukcja snu delta) bya niejednolita midzy laboratoriami
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