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Disturbance of insulin signaling in diabetic patients might lead to the abnormal expression of Syn, damage to mitochondrial function, increase in mitochondrial oxidative stress, and down-regulation of the PI3K/AKT pathway, which in turn could promote the occurrence and development of PD (62)

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Its broad biological relevance has made GHK-Cu a recurring subject in studies focused on extracellular matrix dynamics, regenerative biology, and cellular communication networks

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