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Many cardiovascular risk factors such as diabetes mellitus, hypertension and hyperlipidemia are demonstrated to stimulate the interaction of Cav-1 and eNOS, reduce nitric oxide production resulting in endothelial dysfunction and subsequently the formation of atherosclerotic lesion 41,42,43

A classification of CD4 + T cells on the basis of the patterns of cytokines that they secrete

Putative protective role of autoantibodies against the insulin-like growth factor-1 receptor in Graves disease: results of a pilot study