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In cancer, however, DDR exerts a paradoxical influence: while repair deficiencies promote mutagenesis and tumorigenesis, restoration or hyperactivation of DDR pathways in established tumors fosters resistance by enabling efficient repair of therapy-induced DNA damage
A metabolic basis for midzonal necrosis of the liver
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The metabolic reprogramming of CAFs thus represents a novel mechanism of immunotherapy resistance that can be targeted using nanoparticle-based approaches (35)
Its therapeutic administration is part of advanced protocols in regenerative medicine, sports medicine, musculoskeletal rehabilitation, and post-surgical recovery
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