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ERK1/2 Activation Extracellular signal-regulated kinases 1 and 2 trigger transcription factors c-Fos and c-Jun when activated by BPC-157

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In contrast to melanomas, which acquire additional driver mutations, nevi usually enter a suppressive state of replicative senescence which is regulated by the tumor suppressor gene CDKN2A via its proteins, p14 and p16, and various transcriptional controls of the cell cycle (30, 31)

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