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Women undergoing hormone replacement therapy may therefore experience amplified responses to MOTS-c, a clinically relevant interaction that warrants monitoring

RSL3 can covalently inactivate GPX4 by binding to selenocysteine in the active site of GPX4 [33]

Reporting summary Further information on research design is available in the Nature Portfolio Reporting Summary linked to this Article

The side effects at this dose are also the most pronounced, with nausea affecting approximately 44 percent of participants in trials

Fibroblast Growth Factor 21 dependent TLR4/MYD88/NF-B signaling activation is involved in lipopolysaccharide-induced acute lung injury