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According to the model presented here based on current inactive and active structures of GLP-1R, the canonical peptide activation pathway involves a major conformational change of the ECD, going from a closed inactive to open active state (Fig
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It is an attempt to describe, honestly and with real citations, what the published literature says
ApoE / knockout mice treated with HSYA and its mimetic liposomes (HA-ML@HSYA NPs) via tail vein injection exhibited decreased methylation levels of the autophagy-related gene Atg13 DNA, increased macrophage autophagy, and inhibited ox-LDL uptake, thereby alleviating atherosclerosis
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